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Prevacid (Lansoprazole)

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Generic Prevacid helps with different acid-related disorders. It treats heartburn by decreasing the amount of acid produced in the stomach. Generic Prevacid can also help cure damage which acid can make to your esophagus. Moreover, Generic Prevacid helps cure duodenal ulcers (ulcers in the first part of the intestine), gastric ulcers (stomach ulcers) and stomach ulcers as a result of continuous use of pain medications called NSAIDs, nonsteroidal anti-inflammatory drugs (aspirin, ibuprofen and naproxen).

Other names for this medication:

Similar Products:
Pepcid, Aciphex, Dexilant, Nexium, Protonix


Also known as:  Lansoprazole.


Generic Prevacid helps with different acid-related disorders.

Generic Prevacid helps with different acid-related disorders. It treats heartburn by decreasing the amount of acid produced in the stomach. Generic Prevacid can also help cure damage which acid can make to your esophagus. Moreover, Generic Prevacid helps cure duodenal ulcers (ulcers in the first part of the intestine), gastric ulcers (stomach ulcers) and stomach ulcers as a result of continuous use of pain medications called NSAIDs, nonsteroidal anti-inflammatory drugs (aspirin, ibuprofen and naproxen).

Prevacid is also known as Lansoprazole, Fenzer, Lan, Helicid, Zoton, Inhibitol, Takepron.

Generic Prevacid helps with different acid-related disorders.

Brand name of Generic Prevacid is Prevacid.


Generic Prevacid is available in 3 forms such as Generic Prevacid Capsules (15 mg, 30 mg), Generic Prevacid SoluTab Orally Disintegrating Tablets and Generic Prevacid for Oral Suspension.

Generic Prevacid Capsules

Generic Prevacid Capsules should be taken before eating. If you can't swallow you can open a capsule and sprinkl into 1/4 cup of tomato, apple or orange juice, or on a tablespoon of applesauce, Ensure pudding, cottage cheese, yogurt or strained pears. Make sure to drink and eat food sprinkled with Generic Prevacid right away and be careful not to crush or chew the capsules or the granules. If you want to sprinkle Generic Prevacid granules, you should only do this in the juices and foods mantioned above.

Generic Prevacid SoluTab Orally Disintegrating Tablets

It is not necessary to take Generic Prevacid SoluTab with water. It is easy to swallow it without any water. Generic Prevacid SoluTab has a strawberry flavor.

Generic Prevacid for Oral Suspension

The contents of a packet should be mixed with two tablespoons of water, stirred and drunk immediately. Generic Prevacid for Oral Suspension should not be given through a feeding tube and should not be mix with other liquids or foods. Generic Prevacid for Oral Suspension has a strawberry flavor. 24 hours can be enough for Generic Prevacid to relieve heartburn and many other symptoms of acid reflux disease.

If you want to achieve most effective results do not stop taking Generic Prevacid suddenly.


If you overdose Generic Prevacid and you don't feel good you should visit your doctor or health care provider immediately.


Store at room temperature between 15 and 30 degrees C (59 and 86 degrees F) away from moisture and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Prevacid are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Do not take Generic Prevacid if you are allergic to Generic Prevacid components.

Be careful with Generic Prevacid if you're pregnant or you plan to have a baby.

Generic Prevacid can be taken by children aged 1-17 for GERD.

Do not take Generic Prevacid if you have creatinine clearance less than 30 mL/min.

Do not use Generic Prevacid if you take cisapride, pimozide, astemizole, terfenadine, ergotamine or dihydroergotamine.

Do not use potassium supplements or salt substitutes.

If you want to achieve most effective results without any side effects it is better to avoid alcohol.

Be very careful when you are driving machine.

Do not stop taking Generic Prevacid suddenly.

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Forty-two patients who had undergone endoscopic mucosal resection (EMR) were studied. Mucosal blood flow was measured by the use of a laser Doppler flowmeter in the surrounding mucosa and at the ulcer margin, before, 1 day, 1 week and 4 weeks after EMR. Helicobacter pylori infection was confirmed by the use of bacterial culture and histology. After EMR, patients were randomly assigned to receive 30 mg lansoprazole (u.i.d; L-regimen) or 30 mg lansoprazole (u.i.d.) with 200 mg cetraxate (q.i.d; LC-regimen) for 4 weeks.

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A total of 732 subjects received VPZ or LPZ. The proportion of healed EO subjects at week 4 was 92.3%, 92.5%, 94.4%, 97.0% and 93.2%, respectively, with VPZ 5, 10, 20 and 40 mg and LPZ 30 mg. All VPZ doses were non-inferior to LPZ when adjusted for baseline LA grades A/B and C/D. Among those with LA grades C/D, the proportions of healed EO subjects were 87.3%, 86.4%, 100%, 96.0% and 87.0%, respectively, with VPZ 5, 10, 20 and 40 mg and LPZ 30 mg. The incidence of adverse events was similar across the groups.

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A new series of N-Substituted 2-(benzhydryl- and benzylsulfinyl)nicotinamides 7 and 8 were synthesized. Upon acid activation in the acidic environment of the parietal cell, these compounds are converted into their active forms, 2,3-dihydro-3-oxoisothiazolo[5,4-b]pyridines 5, which inhibit gastric H+/K(+)-ATPase. Inhibitory activities against [14C]aminopyrine accumulation stimulated by dibutyryl cAMP in isolated rabbit parietal cells in vitro and histamine-induced gastric acid secretion in pylorus-ligated rats by intraduodenal administration in vivo were evaluated, and the structure-activity relationships were examined. Among the compounds synthesized, 2-[(2,4-dimethoxybenzyl)sulfinyl]-N-(4-pyridyl)nicotinamide (8b) showed potent inhibitory activities in vitro and in vivo equivalent to those of omeprazole, a typical H+/K(+)-ATPase inhibitor. Moreover, 8b was much more stable at neutral and weakly acidic pH than omeprazole, lansoprazole, and pantoprazole. Compound 8b is considered to be a promising agent for treating acid-related gastrointestinal disorders.

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H. pylori was resistant to metronidazole in three cases, to clarithromycin in three cases, and to both clarithromycin and metroinidazole in an additional three patients. No resistance to amoxicillin was found. Eradication was obtained in 20 cases (95.2% confidence interval [CI], 76.2-99.9). The only patient in whom infection was not eradicated harbored a metronidazole-resistant (minimum inhibitory concentration > 32 micrograms/ml) strain. No significant side effects were reported.

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This randomized, open-label, comparative five-way crossover study evaluated the 24-h intragastric pH profile of oral esomeprazole 40 mg, lansoprazole 30 mg, omeprazole 20 mg, pantoprazole 40 mg, and rabeprazole 20 mg once daily in 34 Helicobacter pylori-negative patients aged 18-60 yr with symptoms of gastroesophageal reflux disease. Patients were randomly assigned to one of five treatment sequences and study drug was taken on 5 consecutive mornings 30 minutes prior to a standardized breakfast. A washout period of at least 10 days separated each treatment phase.

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To assess the effects of intestinal cytochrome P450 2C19 on the interaction between tacrolimus and proton pump inhibitors, we examined the concentration/dose ratio [(ng/ml)/(mg/day)] of tacrolimus coadministered with omeprazole (20 mg) or lansoprazole (30 mg) to 89 adult living-donor liver transplant patients on postoperative days 22 to 28, considering the CYP2C19 genotypes of the native intestine and the graft liver, separately. The concentration/dose ratio of tacrolimus coadministered with omeprazole was significantly higher in patients with two variants (*2 or *3) for intestinal CYP2C19 (median, 6.38; range, 1.55-22.9) than intestinal wild-type homozygotes (median, 2.11; range, 1.04-2.54) and heterozygotes (median, 2.11; range, 0.52-4.33) (P = 0.010), but the extent of the increase was attenuated by carrying the wild-type allele in the graft liver even when patients were CYP3A5*1 noncarriers. Conversely, the CYP2C19 polymorphisms both in the native intestine and in the graft liver little influenced the interaction between tacrolimus and lansoprazole, but CYP3A5*1 noncarriers showed higher tacrolimus concentration/dose ratio than CYP3A5*1 carriers. Furthermore, our experiments in vitro revealed that lansoprazole had a stronger inhibitory effect on the CYP3A5-mediated metabolism of tacrolimus than omeprazole, although not significantly (IC(50) = 19.9 +/- 13.8 microM for lansoprazole, 53.7 +/- 6.1 microM for omeprazole). Our findings suggest that intestinal and graft liver CYP2C19 plays a relatively greater role in the metabolism of omeprazole than it does for lansoprazole, so that the effects of CYP3A5 on the metabolism of tacrolimus might be masked by the interaction with omeprazole associated with the CYP2C19 genotype.

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The intake of proton pump inhibitors may interfere with the reliability of the urea breath test.

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Helicobacter pylori resistance to antibiotics is the main factor for therapy failure, while other features remain largely unknown. The aims of this study are to investigate the relationship of antibiotic resistance and in vitro internalization activity between cure and failure isolates and to determine whether failures are associated with persistence of the same predominant strain.

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A clinical decision analysis comparing the PPI-first strategy and the H2RA-first "step-up" strategy for the acute treatment of reflux esophagitis in Japan was performed, using a Markov chain approach.

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In group A, maximal concentration (C(max)) was 1023 +/- 775 (mg. L(-1))/(17 mg. m(-2)), time to reach C(max) was 1.8 +/- 0.8 hours, elimination half-life was 1.5 +/- 2.0 hours, area under the concentration-time curve from time zero to infinity [AUC(0-infinity)] was 3503 +/- 6025 (mg. L(-1). h)/(17 mg. m(-2)), apparent plasma clearance was 0.57 +/- 0.47 L. h(-1). kg(-1), and apparent volume of distribution was 0.61 +/- 0.36 L. kg(-1). In group B, C(max) was 750 +/- 511 (mg. L(-1))/(17 mg. m(-2)), time to reach C(max) was 1.8 +/- 1.1 hours, elimination half-life was 1.2 +/- 1.1 hours, AUC(0-infinity) was 2351 +/- 3691 (mg. L(-1). h)/(17 mg. m(-2)), apparent plasma clearance was 0.71 +/- 0.50 L. h(-1). kg(-1), and apparent volume of distribution was 0.9 +/- 0.7 L. kg(-1). No influence of age was shown on pharmacokinetic parameters in both groups. However, data suggested that elimination was reduced in neonates and higher in infants than in adults. The values for 24-hour percentage of time at gastric pH <4 and pH <3 were 61% +/- 21% and 51% +/- 21% (group A) and 47% +/- 24% and 37% +/- 21% (group B), respectively. In both groups the antisecretory effect decreased with age, and in group A it was positively correlated to C(max) and AUC(0-infinity). The mean gastrin serum concentration significantly increased (+31%) after 12.6 +/- 1.5 days of treatment.

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prevacid dosage pediatrics 2015-11-15

Proton-pump Inhibitors exert transient negative effects on H. pylori viability, morphology, and urease test. Accordingly, cessation of PPI consumption buy prevacid at least 12 days before endoscopy could help avoiding false-negative results of H. pylori diagnostic tests.

prevacid recommended dosage 2015-08-22

Proton-pump inhibitor and ranitidine bismuth citrate-based triple regimens buy prevacid are the two recommended first line treatments for the eradication of Helicobacter pylori. We aimed to compare the effectiveness and tolerability of these two treatments in a prospective, multicentric, randomized study.

prevacid 45 mg 2015-09-14

This post hoc analysis suggests that sex, age and severity of typical RE symptoms affect HRQOL in patients with RE in Japan, and such factors do not affect the improvement of HRQOL with LPZ treatment. The present study suggests a positive impact of LPZ; however, it is difficult to estimate the degree of improvement in HRQOL that may have arisen from symptomatic relief as part of the natural history of the disease from the study. Our results may overestimate the efficacy of LPZ. Further well- Motrin Dosing Pediatrics controlled clinical studies are needed to confirm the efficacy of LPZ on HRQOL in patients with RE.

prevacid dose infant 2016-10-22

At 6 months, lansoprazole doses of 60, 90, 120 and 180 mg/d maintained BAO < 10 mmol H+/h in 9, 2, 1 and 1 patient, respectively. No Levitra 100mg Pills significant changes in FSG, endocrine cells densities and biological parameters were noted during treatment. Neither adverse events nor carcinoid tumors were observed. We conclude that lansoprazole is efficient and well tolerated in patients with ZES.

prevacid dose neonate 2017-11-11

To compare the annual cost of proton pump inhibitors (PPIs) in the United States Rulide Paediatric Dose and in the United Kingdom.

prevacid tablets infants 2016-05-13

Proton pump inhibitors (PPIs), which are widely used for the treatment of peptic ulcers and gastroesophageal diseases, reduce both basal and stimulated gastric acid secretion by inhibiting the parietal cell enzyme H(+)-K(+)-adenosine triphosphatase. There have been several reports of hypersensitivity reactions to PPIs but anaphylaxis is very rare. We report on two Arava Cost cases of anaphylaxis to PPIs.

prevacid reviews 2016-08-18

The number of PPI prescriptions decreased during the intervention and was sustained at least three quarters afterward. This low-intensity, Paracetamol Medicine Brands practice-based intervention may serve as a model for other health care systems.

prevacid dissolving tablets 2017-05-10

Neither short nor long treatment with pantoprazole modified the Evista Drug Cost ability of neutrophils to migrate in response to carrageenan or H. pylori. The same results were obtained when omeprazole or lanzoprazole was used. Dexamethasone, alone, a potent anti-inflammatory drug, was able to reduce polymorphonuclear and mononuclear cell migration.

prevacid dose toddler 2015-06-09

We studied the gastric acidity, gastric emptying, and proton pump inhibitor (PPI) plasma levels in 10 patients with PPI-resistant ulcers. The pH3 holding-time ratio was low in nine of these patients, with an average ratio of only 39%. PPI plasma levels in those patients were also much lower than in those with nonresistant ulcers. Gastric emptying, determined by the acetaminophen method, was reduced in all 10 patients. Therefore, PPI-resistant ulcers appear to result from insufficient inhibition of gastric acidity, with reduced gastric emptying interfering with the absorption of PPIs. In patients with mild reductions in gastric emptying, PPI plasma levels increased after a change from single-unit enteric-coated tablets (omeprazole) to multiunit enteric-coated granules in capsules (lansoprazole). This change in formulation markedly inhibited gastric acidity and led to rapid healing. In patients with moderate reductions in gastric emptying, doubling the dose of lansoprazole was effective. In patients with severely reduced gastric emptying, there appeared to be a limit to the Discontinuing Cymbalta Medication effectiveness of oral administration of PPIs. Changing the formulation and doubling the dose to compensate for reduced gastric emptying are effective approaches in the treatment of patients with PPI-resistant ulcers.